By Dial A Vet Veterinary Team
This companion page expands symptoms, causes, and treatment themes for canine Addison’s disease (hypoadrenocorticism). For the primary overview, see Addison’s disease in dogs.
Important: No steroid or mineralocorticoid doses are listed here.
Key takeaways
- Primary Addison’s is usually immune-mediated destruction of adrenal cortex.
- Secondary forms relate to pituitary ACTH problems or abrupt withdrawal of long-term steroids in some contexts.
- Classic lab clues include low sodium, high potassium, and sometimes anemia or kidney-value changes from dehydration.
- “Atypical” presentations can lack classic electrolyte changes and still need endocrine testing.
- Lifelong monitoring is normal after diagnosis.
Symptom patterns owners report
- On-again, off-again GI upset
- Exercise intolerance or shaking
- Increased thirst in some dogs
- Bradycardia or weak pulses noted at the clinic
- Acute crisis after boarding, travel, or illness
Treatment themes (clinic-directed)
- Glucocorticoid replacement for cortisol deficiency
- Mineralocorticoid support when aldosterone is lacking
- Electrolyte monitoring and dose adjustments only under veterinary supervision
- Extra glucocorticoid coverage plans for stressful events when your veterinarian advises them
Do not borrow another pet’s adrenal medications or change intervals from online charts.
FAQ
Is Addison’s the opposite of Cushing’s?
Informally, yes: Addison’s is too little cortisol activity; Cushing’s is too much. Testing and therapy differ completely.
Can diet alone treat Addison’s?
No. Hormone replacement is medical therapy supervised by a veterinarian.
Talk with a vet
Crisis signs: go now. Management questions when stable: dialavet.com.
References
- Merck Veterinary Manual. Hypoadrenocorticism.
- Veterinary endocrinology textbooks and ACVIM resources on adrenal disease.
Disclaimer: Educational only. No doses or DIY replacement schedules are provided.
